When working with Nimodipine, a selective calcium channel blocker that helps keep brain arteries open after a bleed. Also known as Nimodipine HCl, it is commonly prescribed after a subarachnoid hemorrhage, a type of brain bleed that often triggers cerebral vasospasm. The drug belongs to the class of calcium channel blockers, which relax smooth muscle in blood vessels and improve blood flow. By targeting these vessels, nimodipine can reduce the risk of delayed ischemic neurological deficits, a major concern after a bleed.
Because nimodipine works directly on the brain’s circulation, its dosing schedule is strict: the typical adult regimen is 60 mg every four hours for 21 days. Missed doses can lower effectiveness, so setting alarms helps stay on track. Common side‑effects include mild dizziness, low blood pressure, and headache, but most patients tolerate the medication well when taken with food. It’s important to avoid grapefruit juice, which can raise drug levels and increase side‑effects. If you’re looking for reliable info on nimodipine dosage, safety checks and drug interactions, the articles below cover everything from basic pharmacology to real‑world patient tips.
Below you’ll find a curated collection of posts that dive deeper into nimodipine’s role in managing subarachnoid hemorrhage, compare it with other calcium channel blockers, and offer practical advice on monitoring and side‑effect management. Explore the range of content to get a full picture of how this medication fits into stroke prevention and neurological care.
Learn how medications like nimodipine, antihypertensives, and antiepileptics manage subarachnoid hemorrhage symptoms, prevent complications, and improve recovery outcomes.